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Angela Gupta


Mary Baldwin College, B.S. in Biology (2012)
Hometown: Lynchburg, VA
PhD Advisor: Tomasz Kordula, PhD
Graduate Program: Biochemistry and Molecular Biology


Research Interests:

Angela’s dissertation focuses on investigating the role of the transcription factor RelB in controlling cytokine production during neuroinflammation and cancer. Additionally, Angela has worked on various research projects centered around cytokine biology and sphingolipid signaling. Because of her strong interest in better understanding inflammatory disease, Angela is also involved in various clinical research projects studying the pathogenesis and epidemiology of sterile inflammatory skin disorders.

Personal Bio:

Angela grew up in Lynchburg, VA and graduated from Mary Baldwin College in 2012 summa cum laude with her degree in Biology. In her free time Angela enjoys spending time with friends and family, being outdoors, and trying out new restaurants.

Honors, Awards, and Grants:

Ruth L. Kirschstein National Research Service Award (NRSA) Research Fellowship 
    National Institutes of Health- National Cancer Institute 
    Cytokine-induced RelB phosphorylation in glioblastoma multiforme 
    Principal Investigator 
    F30 CA203447-01A1
VCU School of Medicine, Herbert John Evans Jr. Award (2017)
Alpha Epsilon Lambda (2017)
VCU School of Medicine, C. C. Clayton Award (2016)
Phi Kappa Phi (2015)
Alpha Omega Alpha (2014)

Professional, Leadership, and Service Activities:

MD-PhD Student Council representative (2017-current)
VCU Department of Biochemistry Student Seminar Organizer (2015-2016)


Gupta, A.S., Ortega-Loayza A.G. (in press) Ocular pyoderma gangrenosum: A systematic review. J Am Acad Dermatol.

Gupta, A.S., O’Connor P.W., Ortega-Loayza A.G. Vulvar edema and weight loss in a pediatric patient. J Am Acad Dermatol. 2015 Dec;73(6):e193-4.

Yester, J.Y., Bryan, L., Waters, M.W., Mierzenski, B., Biswas, D.B., Gupta, A.S., Bhardwaj, R., Surace, M.J., Eltit, J.M., Milstien, S., Spiegel, S., Kordula, T.K. S1P inhibits IL-1 induced expression of CCL5 via c-fos dependent suppression of interferon-β amplification loop. The FASEB Journal (2015): fj-15.